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SPASMODIC DYSPHONIA
SD is a focal dystonia characterized by involuntary spasms in the
muscles for voicing (PSA, TA).
Facts:
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Cause is unknown
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Onset is usually gradual
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Average age of onset is between 30 and
50
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More common in females than in males
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Some cases are hereditary (gene on
chromosome 9)
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Often diagnosed following respiratory
tract infections, laryngeal damage due to injury, and vocal overuse
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Symptoms worsen under stressful
conditions and while talking on the phone
Two main categorizations: Adductor, abductor
Adductor:
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Most common for
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Involuntary muscle spasms cause the
vocal folds to slam togethe
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Stiffness of vocal folds
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Tight, strained, strangled or “over
pressurized” voice (Stemple, 2000)
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Prolongation of vowel sounds
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Words are cut off or difficult to
initiate due to spasms
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Stuttering like symptoms
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Most evident in vowels, liquids, glides
Abductor:
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Difficult to diagnose and to treat
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Involuntary muscle spasms in the PCA
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Abrupt, discontinuous escapes of air
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Inability of the TVF to close for
voicing results in a whispered voice quality
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Voiceless consonants are prolonged
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/s/, /h/, /k/ before open vowel sound
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Pitch elevation on initiation of vowels
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Difficulty coordinating speaking and
breathing
TX options:
Successful Voice Therapy:
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Must introduce acquisition of new
voicing skills and patterns not characterized by overpressure and
interruptions
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Eliminate negative effects of surgery
(paralysis) and Botox
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Produce phonation with higher pitch,
increased breathiness, decreased intensity
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Intensive pre-TX therapy can greatly
improve post-TX therapy outcomes
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Therapy goal: reduction of main
components responsible for ADD/SD symptoms: TVF collision force, TVF
contact area, and elevated subglottic air pressures (Ps)
http://www.voicedoctor.net/therapy/dystonia.htm http://www.nidcd.nih.gov/health/voice/spasdysp.asp
http://www.dysphonia.org/spasmodic/
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